A Neurological Model of Dyslexia and Other Domain-specific Developmental Disorders with an Associated Sensorimotor Syndrome.

Abstract

Given mounting evidence that auditory, visual and/or motor dysfunction do not cause developmental dyslexia, but are often associated with it, the present paper proposes a new neurological model of dyslexia which explains how a specific phonological deficit might arise, and sometimes occur together with a more general sensorimotor syndrome. Based on a comprehensive review of the neurology of dyslexia, the model specifies that: 1) Genetically determined focal cortical anomalies in specific left perisylvian language areas are the underlying cause of the phonological deficit; 2) This phonological deficit is the primary cause of reading impairment; 3) Under certain hormonal conditions during gestation, these cortical anomalies induce secondary disruption in sensory pathways, notably in the thalamus. 4) When this happens, the individual affected displays one or several components of a sensorimotor syndrome, which may in some cases aggravate the reading impairment.

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